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Conclusions This narrative review demonstrates that semaglutide’s oral adverse effects cannot yet be explained by direct salivary-gland data, but the convergence of three mechanistic domains—albumin-mediated continuous receptor stimulation, bias toward sustained Gs/cAMP signaling, and β-arrestin–dependent desensitization—provides a coherent hypothesis for salivary hypofunction
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GLP-1RAs also reduce food intake by modulating appetite-regulating pathways in the brain, resulting in weight loss (Koide et al., 2025) (see Figure 1)
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